Search Criteria: Strain Type is "Chromosome Aberration"
| Stock Number |
Strain Name Phenotype |
Standard Supply |
| 000726 | RBF/DnJ | Level 4 |
| The RBF inbred strain arose from crosses with wild mice, originally known as "tobacco mouse", captured in Valle di Poschiavo in S.E. Switzerland. The wild mice originally known as 'tobacco mouse' because of the coat colour. The strain was transferred to Dr. M. Davisson (Dn) in 1981 and subsequently to the production colony of The Jackson Laborotory (J). Mice are homozygous for Robertsonian translocation Rb(1.3)1Bnr, Rb(8.12)5Bnr and Rb(9.14)6Bnr. This strain is useful for production of antibody producing hybridomas. | ||
| 006790 | B6;129P2-Del(14)3Mom/MomJ | Repository- Live |
| The H element was identified by the high homoology of this non-coding DNA between mouse and human. In hemizygotes, the Del(14)3Mom deletion of the H element abolishes expression of Olfr1507 (MOR28) and Olfr1508 (MOR10) genes in cis from the 129P2/OlaHsd chromosome, while expression of Olfr1507 and Olfr1508 genes from the wildtype C57BL/6J chromosome persists. In homozygotes, Olfr1507, Olfr1508, and Olfr1509 (MOR83) genes are not expressed, and there is a graded, distance-related decrease in the numbers of cells expressing more distal olfactory receptor genes, Olfr1510 (MOR29A), Olfr1511 (MOR29B), Olfr1512 (MOR30A), and Olfr1513 (MOR30B). The expression of other olfactory receptor genes on Chromosome 14 such as Olfr1514 (MOR205-1), Olfr749 ((S1), and Olfr221 (MOR205-1) is not affected in an obvious way by the Del(14)3Mom deletion. Expression of olfactory receptor genes loc
..... For more information please see the full phenotype on the strain data sheet | ||
| 001924 | B6EiC3Sn a/A-Ts(1716)65Dn | Repository- Live |
| Segmentally trisomic Ts(1716)65Dn mice provide a postnatal model for Down syndrome. Ts65Dn mice have three copies of most of the genes on mouse Chr 16 that are homologues of human Chr 21 genes. These extra genes, along with the centromere and about 5% of proximal Chr 17 are contained in a small extra chromosome derived from a reciprocal translocation. Neural cognitive deficits and behavioral abnormalities have been noted in Ts65Dn mice. They have spatial learning and memory defects as assessed in the Morris water maze and the radial arm maze, show developmental delay in sensorimotor milestones, and exhibit locomotor hyperactivity, lack of behavioral inhibition, and stereotypic behavior. They perform similar to controls in visual placing, balance, prehensile reflex and traction on a horizontal bar, motor coordination, swimming ability, olfaction orienting. They also show altered noradrenergic transmission in the hippocampus and cerebral cortex and degeneration of basal forebr
..... For more information please see the full phenotype on the strain data sheet | ||
| 001923 | B6EiC3Sn a/A-Ts(417)2Lws Tim/J | Repository- Live |
| The Tim mutation (translocation induced circling mutation) was induced by ethylene oxide in a mutagenesis experiment. The mutation was induced simultaneously with a reciprocal translocation between Chrs 4 and 17. Because the mutation has so far been inseparable from the translocation, it may be that one of the translocation breakpoints has disrupted a gene on Chr 4 or 17. Heterozygotes exhibit circling, head tossing, poor limb coordination, and corneal clouding. The affected mice are usually smaller than wildtype littermates. Inner ear studies have not been done. The mutants swim poorly but can orient themselves in the water. One mutated copy of the gene is dominant over two normal alleles because segmentally trisomic mice with two normal Chrs 4 and 17 and the small 417 translocation product have the behavioral abnormalitites. Breeding is generally poor. | ||
| 000655 | CBA/CaH-T(14;15)6Ca/J | Repository- Live |
| This strain carries the T(14;15)6Ca translocation which results in nondisjunction at a rate of 4.4% in males and 22.2% in females. CBA/CaH-T6 mice have been used in tandem with the CBA/H strain in foreign body tumorigenesis studies in which the T6 chromosome was used as a marker to distinguish donor cells from host. | ||
| 001802 | CByJ.RBF-Rb(8.12)5Bnr/J | Repository- Live |
| Hybridomas are produced by fusing mouse meloma cells that are deficient in adenosine phosphoribosyltransferase (APRT) with spleen cells from mice immunized with specific antigens. A selection system using AAT (adenosine, aminopterin, and thymidine) medium eliminates all but the fusion cells that are APRT+ . Successful hybridomas must also retain the Igh heavy chain locus on chromosome 12 and one of the Igh light chain loci. The Robertsonian chromosome Rb(8.12)5Bnr can be used to produce hybridomas at a high frequency. Selecting for the Aprt locus on chromosome 8 also selects for the Igh locus on chromosome 12 because the two loci are "genetically linked" by the Robertsonian fusion chromosome. This chromosomal aberration congenic was created by backcrossing the Robertsonian chromosome Rb(8.12)5Bnr on the BALB/cByJ genetic background by Dr. Muriel Davisson at The Jackson Laboratory. | ||
| 000874 | RBA/DnJ | Repository- Live |
| 001032 | STOCK In(1)38Rk/J | Repository- Live |
| 001379 | STOCK In(4)56Rk Rd4/J | Repository- Live |
| 000729 | STOCK Rb(11.13)4Bnr/J | Repository- Live |
| 000603 | STOCK T(9;17)138Ca/J | Repository- Live |
| 000929 | TIRANO/EiJ | Repository- Live |
| This wild-derived inbred strain is homozygous for 8 Robertsonian translocations: Rb(1.3)1Bnr, Rb(4.6)2Bnr, Rb(5.15)3Bnr, Rb(11.13)4Bnr, Rb(9.14)6Bnr, Rb(16.17)7Bnr, Rb(2.8)2Lub and Rb(10.12)5Lub (abbreviated Rb1Bnr - Rb7Bnr, Rb2Lub and Rb5Lub, respectively)(LL Washburn, pers. comm.). | ||
| 001392 | ZALENDE/EiJ | Repository- Live |
| This wild-derived inbred strain is homozygous for 7 Robertsonian translocations: Rb(1.3)1Bnr, Rb(4.6)2Bnr, Rb(5.15)3Bnr, Rb(11.13)4Bnr, Rb(8.12)5Bnr, Rb(9.14)6Bnr, and Rb(16.17)7Bnr (abbreviated Rb1Bnr - Rb7Bnr, respectively)(LL Washburn, pers. comm.). | ||
| 006653 | 129S-Del(6)1Mom/MomJ | Repository-Cryopreserved |
| Using chromosome engineering technology, an approximately 600 kilobase genomic region was deleted that contains a cluster of 16 intact V1r vomeronasal receptor genes. The mutant mice display deficits in a subset of VNO-dependent behaviors: the expression of male sexual behavior and maternal aggression is substantially altered. Electrophysiologically, the epithelium of the VNO of such mice does not respond detectably to specific pheromonal ligands. | ||
| 000612 | AEJ.Cg-T(10;14)8Rk/J | Repository-Cryopreserved |
| 001138 | AEJ.Cg-T(3;12)30Rk/J | Repository-Cryopreserved |
| 001102 | AEJ.Cg-T(5;8)3Rk/J | Repository-Cryopreserved |
| 001099 | AEJ/GnRk-Rb(11.14)1Dn/J | Repository-Cryopreserved |
| 001538 | B6 x B6C3Sn a/A-T(1;9)27H/J | Repository-Cryopreserved |
| 000916 | B6 x B6C3Sn a/A-T(5;12)31H/J | Repository-Cryopreserved |
| 000602 | B6 x B6C3Sn a/A-T(8;16)17H/J | Repository-Cryopreserved |
| 000593 | B6 x B6CBCa Aw-J/A-Grid2Lc T(2;6)7Ca MitfMi-wh/J | Repository-Cryopreserved |
| Mice homozygous for the MitfMi spontaneous mutation are characterized by decreased macrophage chemotactic responses, impaired proliferative responses to B cell and T cell mitogens, diminished responses in vitro to T-dependent and T-independent antigens and reduced NK cell activity. | ||
| 000599 | B6 x B6CBCa Aw-J/A-T(5;13)264Ca KitW-v/J | Repository-Cryopreserved |
| 002083 | B6 x B6EiC3 a/A-T(7;16)235Dn/J | Repository-Cryopreserved |
| 003759 | B6 x B6EiC3Sn a/A-T(10;16)232Dn/J | Repository-Cryopreserved |
| 002071 | B6 x B6EiC3Sn a/A-T(11;17)202Dn/J | Repository-Cryopreserved |
| 002113 | B6 x B6EiC3Sn a/A-T(11A2;16B3)238Dn/J | Repository-Cryopreserved |
| 002068 | B6 x B6EiC3Sn a/A-T(11B1;16B5)233Dn/J | Repository-Cryopreserved |
| 002069 | B6 x B6EiC3Sn a/A-T(14E4or5;16B5)225Dn/J | Repository-Cryopreserved |
| 001926 | B6 x B6EiC3Sn a/A-T(15;16)198Dn/J | Repository-Cryopreserved |
| 001832 | B6 x B6EiC3Sn a/A-T(15E;16B1)60Dn/J | Repository-Cryopreserved |
| 003758 | B6 x B6EiC3Sn a/A-T(16C3-4;17A2)65Dn/J | Repository-Cryopreserved |
| 001833 | B6 x B6EiC3Sn a/A-T(1C2;16C3)45Dn/J | Repository-Cryopreserved |
| 001903 | B6 x B6EiC3Sn a/A-T(6F;18C)57Dn/J | Repository-Cryopreserved |
| 001535 | B6 x B6EiC3Sn a/A-T(8A4;12D1)69Dn/J | Repository-Cryopreserved |
| 001831 | B6 x B6EiC3Sn a/A-T(8C3;16B5)164Dn/J | Repository-Cryopreserved |
| 002545 | B6 x RBA/DnJ | Repository-Cryopreserved |
| 000601 | B6 x STOCK a/a T(7;18)50H/J | Repository-Cryopreserved |
| 001962 | B6 x STOCK In(10)17Rk/J | Repository-Cryopreserved |
| 002022 | B6 x STOCK In(2)58Rk/J | Repository-Cryopreserved |
| 000610 | B6 x STOCK Rb(6.15)1Ald/J | Repository-Cryopreserved |
| 000951 | B6 x STOCK T(10;18)18H/J | Repository-Cryopreserved |
| 000597 | B6 x STOCK T(2;16)28H/J | Repository-Cryopreserved |
| 000961 | B6 x STOCK T(2;3)24H/J | Repository-Cryopreserved |
| 000592 | B6 x STOCK T(2;4)13H a/J | Repository-Cryopreserved |
| 000591 | B6 x STOCK T(2;4)1Ca/J | Repository-Cryopreserved |
| 000950 | B6 x STOCK T(2;8)26H/J | Repository-Cryopreserved |
| 000595 | B6 x STOCK T(2;9)11H/J | Repository-Cryopreserved |
| 001820 | B6 x STOCK T(2D;11B5)4Dn/J | Repository-Cryopreserved |
| 002016 | B6(Cg)-Aw-J EdaTa-6J Chr YB6-Sxr/EiJ | Repository-Cryopreserved |
| 000552 | B6-Aw-J-EdaTa-6J.Cg-Sxr | Repository-Cryopreserved |
| 001730 | B6-Aw-J-EdaTa-6J.Cg-Sxrb Hya-/J | Repository-Cryopreserved |
| 000600 | B6-Gpi1b x B6CBCa Aw-J/A-T(7;15)9H Gpi1a/J | Repository-Cryopreserved |
| 005383 | B6.129S6-Dp(16Cbr1-ORF9)1Rhr/J | Repository-Cryopreserved |
| These mice are trisomic for the mouse chromosome segment orthologus to the human chromosome 21 Down syndrome critical region (DSCR). The borders of the 3.9 Mb region are defined by the carbonyl reductase 1 gene and a site adjacent to the myxovirus (influenza virus) resistance 2 locus. Trisomic mice are viable, fertile and are significantly larger than euploid littermates. Mandible size is enlarged as is skull size, with an overall elongation of the rostrocaudal aspect in comparison to euploid littermates. This mouse may be useful in studies exploring the consequences of polyploidy involving the DSCR. | ||
| 005535 | B6.129S7-Del(11Cops3-Zfp179)1Jrl/J | Repository-Cryopreserved |
| These mutant mice possess an engineered deletion spanning approximately 3 Mb on mouse Chromosome 11. The region involved encompasses a chromosomal segement that shares conserved synteny with the Smith-Magenis syndrome (SMS) critical interval on human Chromosome 17. Mice carrying one copy of the deletion prove to be viable while mice homozygous for the deletion are embryonic lethal. Heterozygous males suffer from reduced fertility, exhibiting reduced sperm counts and an increase in sperm structural abnormalities. Mutant mice weigh less than their wild type littermates at birth but rapidly gain weight such that by 4 months of age, they exceed wild type weight and eventually become obese (60 grams by 8 months of age). Mutant mice exhibit craniofacial abnormalities characterized by overall shorter skulls with broader, shorter snouts and nasal bones. Mutants also produce abnormal electroencephalograms (EEG) with tonic clonic-type seizures being observed in 22% of the mice tested. Behavioral
..... For more information please see the full phenotype on the strain data sheet | ||
| 005536 | B6.129S7-Dp(11Cops3-Zfp179)1Jrl/J | Repository-Cryopreserved |
| Homozygous mutant mice are viable and fertile. Studies employing heterozygous mutant mice (where the duplication exists only on one chromosome) indicate that mutant mice weigh less than their wildtype litter mates. Spleens are noted to be slightly smaller than those from wild type mice. Mutant mice also exhibit impaired contextual fear conditioning. Male heterozygotes have been shown to be hyperactive in open field analysis. This mutant mouse may be useful in studies exploring the consequences of duplications involving the SMS critical interval. (Mice bearing the reciprocal deficiency mutation are also available; see Stock: 005535) | ||
| 001914 | B6.Cg-Is(HSR;1)1Lub/J | Repository-Cryopreserved |
| 000861 | B6.Cg-Rb(1.3)1Lub/J | Repository-Cryopreserved |
| 000952 | B6.Cg-Rb(10.11)8Bnr/J | Repository-Cryopreserved |
| 000960 | B6.Cg-Rb(2.8)2Lub/J | Repository-Cryopreserved |
| 001733 | B6.WMP-Rb(10.17)9Mpl/J | Repository-Cryopreserved |
| 001734 | B6.WMP-Rb(13.15)10Mpl/J | Repository-Cryopreserved |
| 001727 | B6.WMP-Rb(2.16)3Mpl/J | Repository-Cryopreserved |
| 001728 | B6.WMP-Rb(3.12)4Mpl/J | Repository-Cryopreserved |
| 001729 | B6.WMP-Rb(4.6)5Mpl/J | Repository-Cryopreserved |
| 001732 | B6.WMP-Rb(8.9)8Mpl/J | Repository-Cryopreserved |
| 001378 | B6;D2-In(3)55Rk Uoxin/J | Repository-Cryopreserved |
| The Uoxin homozygous phenotype has incomplete penetrance. While 63% of Uoxin homozygotes die by 12-14 days of age, those that live to adulthood generally live a normal breeding life span. Homozygous adults display chronic polyuria, increased serum BUN and creatinine levels, and hydronephrosis with a concomitant inflammatory response that is followed by glomerular and tubular dilation. (Cook et al., 2001.) | ||
| 000604 | B6C3 a/A-T(10;13)199H +/+ Lystbg-J/J or Lystbg-2J/J | Repository-Cryopreserved |
| 001752 | B6CBCa Aw-J/A-T(7;15)9H/J | Repository-Cryopreserved |
| 001796 | B6Ei.Cg-Rb(1.2)18Lub/J | Repository-Cryopreserved |
| 001921 | B6Ei.Cg-Rb(1.7)1Rma/J | Repository-Cryopreserved |
| 001386 | B6Ei.Cg-Rb(10.12)5Lub/J | Repository-Cryopreserved |
| 001617 | B6Ei.Cg-Rb(10.15)23Lub/J | Repository-Cryopreserved |
| 001795 | B6Ei.Cg-Rb(11.13)4Bnr/J | Repository-Cryopreserved |
| 000150 | B6Ei.Cg-Rb(11.16)2H/J | Repository-Cryopreserved |
| 000906 | B6Ei.Cg-Rb(16.17)8Lub-tw3Lub/+ +/J | Repository-Cryopreserved |
| 001387 | B6Ei.Cg-Rb(2.17)11Rma/J | Repository-Cryopreserved |
| 001613 | B6Ei.Cg-Rb(5.14)21Lub/J | Repository-Cryopreserved |
| 001616 | B6Ei.Cg-Rb(5.15)4Lub/J | Repository-Cryopreserved |
| 001791 | B6Ei.Cg-Rb(5.19)1Wh/J | Repository-Cryopreserved |
| 000762 | B6Ei.Cg-Rb(6.15)1Ald/J | Repository-Cryopreserved |
| 001798 | B6Ei.Cg-Rb(6.16)24Lub/J | Repository-Cryopreserved |
| 001389 | B6Ei.Cg-Rb(6.7)13Rma/J | Repository-Cryopreserved |
| 001615 | B6Ei.Cg-Rb(7.18)9Lub/J | Repository-Cryopreserved |
| 001797 | B6Ei.Cg-Rb(8.12)22Lub/J | Repository-Cryopreserved |
| 000888 | B6Ei.Cg-Rb(8.14)16Rma/J | Repository-Cryopreserved |
| 001792 | B6Ei.Cg-Rb(9.19)163H/J | Repository-Cryopreserved |
| 002021 | B6Ei.LT-Y*/EiJ | Repository-Cryopreserved |
| 001794 | B6Ei.WMP-Rb(13.16)1Mpl/J | Repository-Cryopreserved |
| 002470 | B6EiC3Sn x B6-Rb(5.8)3Dn/J | Repository-Cryopreserved |
| 004850 | B6EiC3Sn-Rb(12.T171665Dn)2Cje/CjeDn | Repository-Cryopreserved |
| Like B6EiC3Sn a/A - Ts(1716)65Dn (Stock No. 001924), this strain has three copies of most of the genes on mouse Chromosome 16 that are homologues of human Chromosome 21 genes. Transmission of the chromosome 16 segmental trisomy through the female germline is significantly improved over Ts65Dn (43% versus 24%). Dendritic spines on granule cells in the fascia dentata are enlarged in size and decreased in density (Villar AJ, et al. 2005). Unlike Ts65Dn, males are fertile. Mice are 20% smaller in size than controls. This strain serves a model for Down syndrome. | ||
| 004861 | B6EiC3Sn-Ts(16C-tel)1Cje | Repository-Cryopreserved |
| Ts(16C-tel)1Cje trisomic mice display decreased exploratory behavior and have spatial learning deficits detectable in the hidden platform and probe versions of the Morris water maze. These learning deficits are less severe than those of mice carrying Ts(1716)Dn, a larger distal chromosome 16 translocation. The degeneration of basal forebrain cholinergic neurons that is found in Ts(1716)Dn carriers has not been found by six months of age in mice carrying Ts(16C-tel)1Cje. Additionally, the male sterility found in Ts(1716)Dn carriers is not a phenotype associated with Ts(16C-tel)1Cje. (Sago et al., 1998.) Despite having fewer trisomic genes, Ts(16C-tel)1Cje carriers display much of the same craniofacial dysmorphology as that found in Ts(1716)Dn, which includes brachycephaly, reduced interorbital breadth, and smaller mandible. (Richtsmeier et al., 2002.) | ||
| 002460 | C3H/HeJ-Oca2p-J Is(7;1)40H/J | Repository-Cryopreserved |
| 000975 | C57BL/6J x STOCK Rb(1.3)1Ei/J | Repository-Cryopreserved |
| 000584 | C57BL/6J-+ T(1;2)5Ca/a +/J | Repository-Cryopreserved |
| 001391 | C57BL/6J-Rb(4.15)1Rk/J | Repository-Cryopreserved |
| 001390 | C57BL/6J-Rb(6.18)2Dn/J | Repository-Cryopreserved |
| 000586 | C57BL/6J-T(1;13)70H/J | Repository-Cryopreserved |
| 001961 | C57BL/6JEi x STOCK T T(16;17)43H/+ T(16;17)43H/Ei | Repository-Cryopreserved |
| 001793 | C57BL/6JEi-Rb(X.2)2Ad/J | Repository-Cryopreserved |
| 000714 | CByJ.RBF-Rb(8.12)5Bnr | Repository-Cryopreserved |
| 002040 | RB126Bnr/EiJ | Repository-Cryopreserved |
| 002041 | RB16Bnr/EiJ | Repository-Cryopreserved |
| 000609 | RBB/DnJ | Repository-Cryopreserved |
| 000889 | RBC/DnJ | Repository-Cryopreserved |
| 001000 | RBD/DnJ | Repository-Cryopreserved |
| 000807 | RBJ/DnJ | Repository-Cryopreserved |
| 000851 | STOCK In(1)12Rk/J | Repository-Cryopreserved |
| 000716 | STOCK In(1)1Rk/J | Repository-Cryopreserved |
| 000943 | STOCK In(1)23Rk/J | Repository-Cryopreserved |
| 001911 | STOCK In(1)24Rk T(In1;13)2Rk/J | Repository-Cryopreserved |
| 000946 | STOCK In(1)24Rk/J | Repository-Cryopreserved |
| 000730 | STOCK In(1)46Rk/J | Repository-Cryopreserved |
| 001031 | STOCK In(1)49Rk/J | Repository-Cryopreserved |
| 000724 | STOCK In(10)42Rk/J | Repository-Cryopreserved |
| 001635 | STOCK In(10)57Rk/J | Repository-Cryopreserved |
| 000719 | STOCK In(10)6Rk/J | Repository-Cryopreserved |
| 000834 | STOCK In(11)20Rk/J | Repository-Cryopreserved |
| 000982 | STOCK In(12)25Rk/J | Repository-Cryopreserved |
| 000890 | STOCK In(13)31Rk/J | Repository-Cryopreserved |
| 000727 | STOCK In(13)43Rk/J | Repository-Cryopreserved |
| 001375 | STOCK In(13)50Rk/J | Repository-Cryopreserved |
| 000931 | STOCK In(14)22Rk/J | Repository-Cryopreserved |
| 001024 | STOCK In(14)36Rk/J | Repository-Cryopreserved |
| 001377 | STOCK In(14)54Rk/J | Repository-Cryopreserved |
| 000620 | STOCK In(15)18Rk/J | Repository-Cryopreserved |
| 000920 | STOCK In(15)21Rk/J | Repository-Cryopreserved |
| Heterozygote In(15)21Rk/J mice are phenotypically normal. The inversion is embryonic lethal in homozygotes. Chromosomal inversions may be modified for use as balancer chromosomes in mutagenesis screens. | ||
| 001023 | STOCK In(15)35Rk/J | Repository-Cryopreserved |
| 001376 | STOCK In(16)51Rk/J | Repository-Cryopreserved |
| 000721 | STOCK In(19)37Rk/J | Repository-Cryopreserved |
| 000830 | STOCK In(2)19Rk/J | Repository-Cryopreserved |
| 001637 | STOCK In(2)3Dn/J | Repository-Cryopreserved |
| 000723 | STOCK In(2)40Rk/J | Repository-Cryopreserved |
| 000715 | STOCK In(2)5Rk/J | Repository-Cryopreserved |
| 000850 | STOCK In(3)11Rk/J | Repository-Cryopreserved |
| 001564 | STOCK In(4)1Dn/J | Repository-Cryopreserved |
| 000877 | STOCK In(4)28Rk/J | Repository-Cryopreserved |
| 000897 | STOCK In(4)32Rk/J | Repository-Cryopreserved |
| 000717 | STOCK In(5)2Rk/J | Repository-Cryopreserved |
| 000852 | STOCK In(5)30Rk/J | Repository-Cryopreserved |
| 000907 | STOCK In(5)33Rk/J | Repository-Cryopreserved |
| 000718 | STOCK In(5)9Rk/J | Repository-Cryopreserved |
| 001029 | STOCK In(6)47Rk/J | Repository-Cryopreserved |
| 001030 | STOCK In(6)48Rk/J | Repository-Cryopreserved |
| 000853 | STOCK In(7)13Rk/J | Repository-Cryopreserved |
| 000854 | STOCK In(8)14Rk/J | Repository-Cryopreserved |
| 001020 | STOCK In(8)34Rk/J | Repository-Cryopreserved |
| 000855 | STOCK In(9)26Rk/J | Repository-Cryopreserved |
| 000722 | STOCK In(9)39Rk/J | Repository-Cryopreserved |
| 000919 | STOCK In(X)1H/J | Repository-Cryopreserved |
| 000862 | STOCK Rb(1.7)1Rma/J | Repository-Cryopreserved |
| 000891 | STOCK Rb(10.11)5Rma/J | Repository-Cryopreserved |
| 000614 | STOCK Rb(10.11)8Bnr/J | Repository-Cryopreserved |
| 000892 | STOCK Rb(10.12)5Lub/J | Repository-Cryopreserved |
| 000895 | STOCK Rb(11.13)6Lub/J | Repository-Cryopreserved |
| 001100 | STOCK Rb(13.16)1Mpl/J | Repository-Cryopreserved |
| 000615 | STOCK Rb(16.17)7Bnr | Repository-Cryopreserved |
| Please see Rb(16.17)7Bnr at the Cytogenetics Models Resource for more information on this strain. | ||
| 003304 | STOCK Rb(16.17)7Bnr-Myo15sh2-2J/J | Repository-Cryopreserved |
| 000898 | STOCK Rb(16.17)8Lub/J | Repository-Cryopreserved |
| 000865 | STOCK Rb(2.17)11Rma/J | Repository-Cryopreserved |
| 000896 | STOCK Rb(2.18)6Rma Rb(12.14)8Rma/J | Repository-Cryopreserved |
| 000867 | STOCK Rb(2.18)6Rma/J | Repository-Cryopreserved |
| 000864 | STOCK Rb(2.8)2Lub Rb(7.18)9Lub/J | Repository-Cryopreserved |
| 000871 | STOCK Rb(3.15)2Rk/J | Repository-Cryopreserved |
| 000980 | STOCK Rb(3.8)2Ei/J | Repository-Cryopreserved |
| 000869 | STOCK Rb(3.8)2Rma/J | Repository-Cryopreserved |
| 000870 | STOCK Rb(3.9)12Lub/J | Repository-Cryopreserved |
| 000873 | STOCK Rb(4.11)12Rma/J | Repository-Cryopreserved |
| 000876 | STOCK Rb(4.15)4Rma/J | Repository-Cryopreserved |
| 000720 | STOCK Rb(4.6)2Bnr/J | Repository-Cryopreserved |
| 000878 | STOCK Rb(5.14)21Lub/J | Repository-Cryopreserved |
| 000880 | STOCK Rb(5.15)15Rma/J | Repository-Cryopreserved |
| 000881 | STOCK Rb(5.17)7Rma/J | Repository-Cryopreserved |
| 000882 | STOCK Rb(5.19)1Wh/J | Repository-Cryopreserved |
| 001884 | STOCK Rb(5.6)1Ty/J | Repository-Cryopreserved |
| 000885 | STOCK Rb(6.16)24Lub | Repository-Cryopreserved |
| Please see Rb(6.16)24Lub at the Cytogenetics Models Resource for more information on this strain. | ||
| 000887 | STOCK Rb(8.12)22Lub/J | Repository-Cryopreserved |
| 000596 | STOCK T(2;11)30H/+ x AEJ-a Gdf5bp-H/J or A/J-a Gdf5bp-J/J | Repository-Cryopreserved |
| 000970 | STOCK T(2;16)28H A/T(2;16)28H a/J | Repository-Cryopreserved |
| 000590 | STOCK T(2;4)1Sn a/J | Repository-Cryopreserved |
| 000594 | STOCK T(2;8)26H a/T(2;8)26H a Tyrp1+/Tyrp1b/J | Repository-Cryopreserved |
| 001101 | STOCK T(3;4)5Rk Tyrp1b/J | Repository-Cryopreserved |
| 001816 | STOCK T(7;18)50H/J | Repository-Cryopreserved |
| 001628 | STOCK T(9;17)10Ad/J | Repository-Cryopreserved |
| 000583 | STOCK T(X;16)16H +/+ EdaTa | Repository-Cryopreserved |
| 002017 | STOCK Tp(1)1Rk/J | Repository-Cryopreserved |
| 000588 | TF/GnLe-T(1;17)190Ca +/+ tf/J | Repository-Cryopreserved |
| 001746 | WMP/PasDnJ | Repository-Cryopreserved |
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How to Register Interest
Please indicate your interest in purchasing any of the strains listed below when they become available for distribution by checking the box next to the strain(s) of interest and then selecting the "Continue" button which leads to an Interest Form.View a Data sheet for New Strains Under Development
Select the strain name to link to the strain data sheet.
New Strains Under DevelopmentThe Jackson Laboratory serves as a worldwide distributor and national repository for common and rare strains of inbred mice and mice carrying spontaneous mutations or induced mutations (i.e., transgenic, targeted/"knockout", or chemically induced mutations). At any one time, we have over 100 strains at various stages of development and colony expansion. Strains "Under Development" fall into two categories depending on the anticipated demand from the scientific community.
- Receive periodic updates on the status of the colony UNDER DEVELOPMENT
- Obtain advance notification of strain availability and opportunity to order prior to the strain being published as available
- Provide input affecting speed and quantity of availability
It is VERY IMPORTANT that you register interest in strains Under Development. The anticipated demand for a strain enables us to determine effectively the distribution plan for each strain Under Development. Registering interest also provides benefits to you (including advance notification of pending availability). Whether a strain is made available from a live colony OR from our cryopreservation repository, you may want to consider the option of Dedicated Supply. To learn more about Dedicated Supply, go to Services.
- Strains that will be made available from a live distribution colony at The Jackson Laboratory.
These strains are designated as: "Under Development for Distribution Colony"- Strains that will be made available through the Cryopreservation Repository.
These strains are designated as: "Under Development for Cryopreservation Repository"
Send questions to our Technical Support team using the Express Technical Support Form.
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