Search Criteria: Strain Type is "Translocation"

JAX® Mice Strains

Stock
Number
Strain Name
 
Phenotype
Standard Supply
000726 RBF/DnJ
Level 4
The RBF inbred strain arose from crosses with wild mice, originally known as "tobacco mouse", captured in Valle di Poschiavo in S.E. Switzerland. The wild mice originally known as 'tobacco mouse' because of the coat colour. The strain was transferred to Dr. M. Davisson (Dn) in 1981 and subsequently to the production colony of The Jackson Laborotory (J). Mice are homozygous for Robertsonian translocation Rb(1.3)1Bnr, Rb(8.12)5Bnr and Rb(9.14)6Bnr. This strain is useful for production of antibody producing hybridomas.
000655 CBA/CaH-T(14;15)6Ca/J
Repository- Live
This strain carries the T(14;15)6Ca translocation which results in nondisjunction at a rate of 4.4% in males and 22.2% in females. CBA/CaH-T6 mice have been used in tandem with the CBA/H strain in foreign body tumorigenesis studies in which the T6 chromosome was used as a marker to distinguish donor cells from host.
000874 RBA/DnJ
Repository- Live
000603 STOCK T(9;17)138Ca/J
Repository- Live
000929 TIRANO/EiJ
Repository- Live
This wild-derived inbred strain is homozygous for 8 Robertsonian translocations: Rb(1.3)1Bnr, Rb(4.6)2Bnr, Rb(5.15)3Bnr, Rb(11.13)4Bnr, Rb(9.14)6Bnr, Rb(16.17)7Bnr, Rb(2.8)2Lub and Rb(10.12)5Lub (abbreviated Rb1Bnr - Rb7Bnr, Rb2Lub and Rb5Lub, respectively)(LL Washburn, pers. comm.).
001392 ZALENDE/EiJ
Repository- Live
This wild-derived inbred strain is homozygous for 7 Robertsonian translocations: Rb(1.3)1Bnr, Rb(4.6)2Bnr, Rb(5.15)3Bnr, Rb(11.13)4Bnr, Rb(8.12)5Bnr, Rb(9.14)6Bnr, and Rb(16.17)7Bnr (abbreviated Rb1Bnr - Rb7Bnr, respectively)(LL Washburn, pers. comm.).
000612 AEJ.Cg-T(10;14)8Rk/J
Repository-Cryopreserved
001138 AEJ.Cg-T(3;12)30Rk/J
Repository-Cryopreserved
001102 AEJ.Cg-T(5;8)3Rk/J
Repository-Cryopreserved
001538 B6 x B6C3Sn a/A-T(1;9)27H/J
Repository-Cryopreserved
000916 B6 x B6C3Sn a/A-T(5;12)31H/J
Repository-Cryopreserved
000602 B6 x B6C3Sn a/A-T(8;16)17H/J
Repository-Cryopreserved
000593 B6 x B6CBCa Aw-J/A-Grid2Lc T(2;6)7Ca MitfMi-wh/J
Repository-Cryopreserved
Mice homozygous for the MitfMi spontaneous mutation are characterized by decreased macrophage chemotactic responses, impaired proliferative responses to B cell and T cell mitogens, diminished responses in vitro to T-dependent and T-independent antigens and reduced NK cell activity.
000599 B6 x B6CBCa Aw-J/A-T(5;13)264Ca KitW-v/J
Repository-Cryopreserved
002083 B6 x B6EiC3 a/A-T(7;16)235Dn/J
Repository-Cryopreserved
003759 B6 x B6EiC3Sn a/A-T(10;16)232Dn/J
Repository-Cryopreserved
002071 B6 x B6EiC3Sn a/A-T(11;17)202Dn/J
Repository-Cryopreserved
002113 B6 x B6EiC3Sn a/A-T(11A2;16B3)238Dn/J
Repository-Cryopreserved
002068 B6 x B6EiC3Sn a/A-T(11B1;16B5)233Dn/J
Repository-Cryopreserved
002069 B6 x B6EiC3Sn a/A-T(14E4or5;16B5)225Dn/J
Repository-Cryopreserved
001926 B6 x B6EiC3Sn a/A-T(15;16)198Dn/J
Repository-Cryopreserved
001832 B6 x B6EiC3Sn a/A-T(15E;16B1)60Dn/J
Repository-Cryopreserved
003758 B6 x B6EiC3Sn a/A-T(16C3-4;17A2)65Dn/J
Repository-Cryopreserved
001833 B6 x B6EiC3Sn a/A-T(1C2;16C3)45Dn/J
Repository-Cryopreserved
001903 B6 x B6EiC3Sn a/A-T(6F;18C)57Dn/J
Repository-Cryopreserved
001535 B6 x B6EiC3Sn a/A-T(8A4;12D1)69Dn/J
Repository-Cryopreserved
001831 B6 x B6EiC3Sn a/A-T(8C3;16B5)164Dn/J
Repository-Cryopreserved
002545 B6 x RBA/DnJ
Repository-Cryopreserved
000601 B6 x STOCK a/a T(7;18)50H/J
Repository-Cryopreserved
000951 B6 x STOCK T(10;18)18H/J
Repository-Cryopreserved
000597 B6 x STOCK T(2;16)28H/J
Repository-Cryopreserved
000961 B6 x STOCK T(2;3)24H/J
Repository-Cryopreserved
000592 B6 x STOCK T(2;4)13H a/J
Repository-Cryopreserved
000591 B6 x STOCK T(2;4)1Ca/J
Repository-Cryopreserved
000950 B6 x STOCK T(2;8)26H/J
Repository-Cryopreserved
000595 B6 x STOCK T(2;9)11H/J
Repository-Cryopreserved
001820 B6 x STOCK T(2D;11B5)4Dn/J
Repository-Cryopreserved
000600 B6-Gpi1b x B6CBCa Aw-J/A-T(7;15)9H Gpi1a/J
Repository-Cryopreserved
000604 B6C3 a/A-T(10;13)199H +/+ Lystbg-J/J or Lystbg-2J/J
Repository-Cryopreserved
001752 B6CBCa Aw-J/A-T(7;15)9H/J
Repository-Cryopreserved
002021 B6Ei.LT-Y*/EiJ
Repository-Cryopreserved
004861 B6EiC3Sn-Ts(16C-tel)1Cje
Repository-Cryopreserved
Ts(16C-tel)1Cje trisomic mice display decreased exploratory behavior and have spatial learning deficits detectable in the hidden platform and probe versions of the Morris water maze. These learning deficits are less severe than those of mice carrying Ts(1716)Dn, a larger distal chromosome 16 translocation. The degeneration of basal forebrain cholinergic neurons that is found in Ts(1716)Dn carriers has not been found by six months of age in mice carrying Ts(16C-tel)1Cje. Additionally, the male sterility found in Ts(1716)Dn carriers is not a phenotype associated with Ts(16C-tel)1Cje. (Sago et al., 1998.) Despite having fewer trisomic genes, Ts(16C-tel)1Cje carriers display much of the same craniofacial dysmorphology as that found in Ts(1716)Dn, which includes brachycephaly, reduced interorbital breadth, and smaller mandible. (Richtsmeier et al., 2002.)
000584 C57BL/6J-+ T(1;2)5Ca/a +/J
Repository-Cryopreserved
000586 C57BL/6J-T(1;13)70H/J
Repository-Cryopreserved
001961 C57BL/6JEi x STOCK T T(16;17)43H/+ T(16;17)43H/Ei
Repository-Cryopreserved
002040 RB126Bnr/EiJ
Repository-Cryopreserved
002041 RB16Bnr/EiJ
Repository-Cryopreserved
000609 RBB/DnJ
Repository-Cryopreserved
000889 RBC/DnJ
Repository-Cryopreserved
001000 RBD/DnJ
Repository-Cryopreserved
000596 STOCK T(2;11)30H/+ x AEJ-a Gdf5bp-H/J or A/J-a Gdf5bp-J/J
Repository-Cryopreserved
000970 STOCK T(2;16)28H A/T(2;16)28H a/J
Repository-Cryopreserved
000590 STOCK T(2;4)1Sn a/J
Repository-Cryopreserved
000594 STOCK T(2;8)26H a/T(2;8)26H a Tyrp1+/Tyrp1b/J
Repository-Cryopreserved
001101 STOCK T(3;4)5Rk Tyrp1b/J
Repository-Cryopreserved
001816 STOCK T(7;18)50H/J
Repository-Cryopreserved
001628 STOCK T(9;17)10Ad/J
Repository-Cryopreserved
000583 STOCK T(X;16)16H +/+ EdaTa
Repository-Cryopreserved
000588 TF/GnLe-T(1;17)190Ca +/+ tf/J
Repository-Cryopreserved
001746 WMP/PasDnJ
Repository-Cryopreserved

(60 stocks)         Back to Top


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