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Reproductive Biology Research
Strains Under Development for all Reproductive Biology Research models.
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| Allele Symbol | Stock Number | Strain Name Description |
Standard Supply |
| Apobtm1.1Zc | 007682 | B6.129X1-Apobtm1.1Zc/J | Repository- Live |
| These apoB38.9 mutant mice may be useful to study the genetic and molecular mechanism of apoB defects and lipid metabolism/liver fat accumulation, the relationship between hepatic steatosis and insulin resistance, the progression of advanced non-alcoholic fatty liver diseases (NAFLD), and atherosclerosis. In addition, all three congenic apoB38.9 strains (Stock No. 007679, Stock No. 007682, and Stock No. 007683) may be useful in conjunction with other apoB mutant mice, including Stock No. 002053 (apoB70), Stock No. 002876 (apoB48-only), and Stock No. 002877 (apoB100-only). | |||
| Apobtm1.1Zc | 007683 | CByJ.129X1(Cg)-Apobtm1.1Zc/J | Repository- Live |
| These apoB38.9 mutant mice may be useful to study the genetic and molecular mechanism of apoB defects and lipid metabolism/liver fat accumulation, the relationship between hepatic steatosis and insulin resistance, the progression of advanced non-alcoholic fatty liver diseases (NAFLD), and atherosclerosis. In addition, all three congenic apoB38.9 strains (Stock No. 007679, Stock No. 007682, and Stock No. 007683) may be useful in conjunction with other apoB mutant mice, including Stock No. 002053 (apoB70), Stock No. 002876 (apoB48-only), and Stock No. 002877 (apoB100-only). | |||
| Apobtm1.1Zc | 007679 | SWR.129X1(B6)-Apobtm1.1Zc/J | Repository- Live |
| These apoB38.9 mutant mice may be useful to study the genetic and molecular mechanism of apoB defects and lipid metabolism/liver fat accumulation, the relationship between hepatic steatosis and insulin resistance, the progression of advanced non-alcoholic fatty liver diseases (NAFLD), and atherosclerosis. In addition, all three congenic apoB38.9 strains (Stock No. 007679, Stock No. 007682, and Stock No. 007683) may be useful in conjunction with other apoB mutant mice, including Stock No. 002053 (apoB70), Stock No. 002876 (apoB48-only), and Stock No. 002877 (apoB100-only). | |||
| Atp8b3tm1Gar | 007659 | 129S-Atp8b3tm1Gar/J | Repository-Cryopreserved |
| -Sperm morphology and motility are unaffected, but tests show that phosphatidylserine (PS) already exists in the outer membrane leaflet before sperm capacitation. When the zona pellucida (ZP) is removed from eggs, fertilization rates are normal; although when the extracellular matrix is intact, fewer spermatozoa bind tightly or penetrate the ZP, and fewer undergo acrosome reactions. This strain may be useful in studies of male fertility. | |||
| Catsper2tm1Gar | 007656 | B6;129S6-Catsper2tm1Gar/J | Repository-Cryopreserved |
| Homozygous males are completely infertile. Sperm cells fail to acquire hyperactivated motility associated with penetration of the extracellular matrix of the egg. This strain may be useful in studies of male fertility. | |||
| Gusbmps | 006564 | B6(C)-KitW-41J Gusbmps/BrkJ | Repository-Cryopreserved |
| H2 | 007958 | B6.Cg-H2b3/FlaCmwJ | Repository- Live |
| This strain, commonly known as B6.K1, has a slow Ped or Qa2negative phenotype. These mice be used for research in embryonic and post-natal development. | |||
| H2 | 007959 | B6.Cg-H2b4/FlaCmwJ | Repository- Live |
| This strain, commonly known as B6.K2, has a fast Ped or Qa2positive phenotype was developed as a control for the slow Ped B6.K1 (Stock 007958). These mice be used for research in embryonic and post-natal development. | |||
| KitW-19H | 002283 | B6.Cg-KitW-19H/EiJ | Repository-Cryopreserved |
| KitW-41J | 006564 | B6(C)-KitW-41J Gusbmps/BrkJ | Repository-Cryopreserved |
| KitlSl-18H | 002993 | B6.Cg-KitlSl-18H/EiJ | Repository-Cryopreserved |
| Ldlrtm1Her | 006906 | B6.Cg-Lepob Ldlrtm1Her/J | Repository- Live |
| The mice may be useful in studies of diabetes, metabolism, hyperglycemia, atherosclerosis, and hypercholesterolemia. | |||
| Lepob | 006906 | B6.Cg-Lepob Ldlrtm1Her/J | Repository- Live |
| The mice may be useful in studies of diabetes, metabolism, hyperglycemia, atherosclerosis, and hypercholesterolemia. | |||
| Ped | 007958 | B6.Cg-H2b3/FlaCmwJ | Repository- Live |
| This strain, commonly known as B6.K1, has a slow Ped or Qa2negative phenotype. These mice be used for research in embryonic and post-natal development. | |||
| Ped | 007959 | B6.Cg-H2b4/FlaCmwJ | Repository- Live |
| This strain, commonly known as B6.K2, has a fast Ped or Qa2positive phenotype was developed as a control for the slow Ped B6.K1 (Stock 007958). These mice be used for research in embryonic and post-natal development. | |||
| Plekha1Gt(ROSA)82Sor | 007201 | B6;129S4-Plekha1Gt(ROSA)82Sor/J | Repository- Live |
| These Plekha1-mutant mice may be useful in studying cellular signaling in development and adult mice; specifically receptor tyrosine kinases (RTK; such as Ras, MAP kinase, PI3K and those in the platelet-derived growth factor (PDGF) family) and immediate early genes (IEG) induced shortly after RTK activation. Homozygous males are infertile and, after 3 weeks of age, exhibit higher than normal (wildtype) weight gain. | |||
| Smad1tm1Sor | 007608 | B6;129-Smad1tm1Sor/J | Repository- Live |
| Homozygotes for the Smad1tm1Sor (also called Smad1C) allele have an embryonic lethal phenotype and do not survive past ED9.5. This mutant mouse strain may be useful in studies of homeostasis and BMP and MAPK signaling pathways during development and in the adult. | |||
| Smad1tm2Sor | 007613 | B6;129-Smad1tm2Sor/J | Repository- Live |
| Homozygotes for the Smad1tm2Sor (also called Smad1L) allele have abnormal gastic mucosal cell homeostasis and fewer primordial germ cells than wildtype controls. This mutant mouse strain may be useful in studies of stomach development, gastic mucosal homeostasis and BMP and MAPK signaling pathways during development and in the adult. | |||
| SryCB | 001543 | C57BL/6JEi-Chr YCB/EiJ | Repository-Cryopreserved |
| Tg(TRAMP)8247Ng | 008215 | (C57BL/6-Tg(TRAMP)8247Ng/J X FVB/NJ)F1/J | Repository- Live |
| These mice express the TRAMP (Transgenic Adenocarcinoma of Mouse Prostate) transgene and develop progressive forms of prostate cancer with distant site metastasis. An earlier onset of the phenotype is observed in these F1 background mice when compared to mice carrying the transgene on the C57BL/6 background. | |||
| 002602 | C57BL/6JEi-Chr YA/HeJ/EiJ | Repository-Cryopreserved | |
| 001574 | C57BL/6JEi-Chr YWSB/Ei/EiJ | Repository-Cryopreserved | |
(22 stocks)
View Strains Newly Available in other Research Areas:
- Apoptosis Research
- Cancer Research
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