Search Criteria: Research Area is "Neurobiology Research: Channel and Transporter Defects (potassium)"

JAX® Mice Strains

Stock
Number
Strain Name
 
Strain Description
Standard Supply
004200 B6;CBACa Aw-J/A-Npr2cn-2J/J
Repository- Live
Mice carrying the Npr2cn-2J display mutation short thick femurs, round heads, disorganized growth plates, and relatively normal vertebrae; A one month old female exhibited the same phenotype as above but also had splayed ribs.
003009 129-Kcne1tm1Sfh/J
Repository-Cryopreserved
This strain is characterized by inner ear defects with a phenotype similar to shaker and waltzer. There is loss of hair cells and many supporting cells in the organ of Corti as well as in all vestibular end organs. The time course of hair cell degeneration is different for each portion of the inner ear. Heart abnormalities of homozygous mutant mice included a longer QT interval at slow heart rates, a paradoxical shorter QT interval at fast heart rates, and an overall exacerbated QT-heart rate adaptation compared with wild-type controls. This mutant serves as a model for Jervell and Lange-Nielsen disease in which patients suffer cardian arrhythmias and profound bilateral deafness.
005830 B6.129P2-Kcnq2tm1Dgen/J
Repository-Cryopreserved
This targeted mutant was created and characterized by Deltagen, Inc. View phenotypic data developed by Deltagen.
000247 B6CBACa Aw-J/A-Kcnj6wv/J
Repository-Cryopreserved
Mice homozygous for the weaver spontaneous mutation (Kcnj6wv) are recognizable in the second postnatal week by their small size, instability of gait, weakness, and hypotonia. Many homozygous mutant mice die at weaning age, but some survive to adulthood, and females may breed. The cerebellum in homozygous mutants is very small, simple, and almost devoid of granule cells, which degenerate during the second week. Heterozygotes behave normally, but they have a smaller than normal cerebellum with a deficiency of granule cells, some of which fail to migrate into the internal granule layer and remain scattered in the molecular layer. Evidence from cultures of mutant and normal cerebellum show that granule cells of Kcnj6wv/Kcnj6wv and Kcnj6wv/+ mice have gene-dosage dependent abnormalities in morphology and cell behavior. Studies using homozygous weaver/wildtype chimeras indicate that the migration defect of granule cells .....
For more information please see the full descriiption on the strain data sheet
004407 C3H/HeJCrl-Kcnq1vtg-2J/J
Repository-Cryopreserved
Kcnq1vtg-2J homozygotes exhibit circling and head-tossing behavior. Both clickbox and acoustic brainstem response (ABR) testing revealed that they are deaf by 8 weeks of age, while age matched heterozygotes have good hearing. This mutation was shown to be an allele of Kcnq1 by complementation testing with the original vertigo mutation, Kcnq1vtg.
003532 C3HeB.129S7-Kcna1tm1Tem/J
Repository-Cryopreserved
Homozygous null mice display an epileptic phenotype (episodic eye blinking, twitching of vibrissae, forelimb paddling, arrested motion, hyperstartle response) beginning during the third postnatal week. Quantitative RNase protection analysis on brain tissue indicates that in heterozygous mice the Kcna1 transcript is reduced to approximately half that observed in wild-type littermates. No transcripts were detected in homozygous mice. Approximately 50% of the homozygous mice die between the third and fifth weeks of life. Mice surviving this period survive for varying periods, depending on genetic background; congenic C3HeB/FeJ mice usually die at 6-8 weeks; hybrid N:NIHS-BC mice have been maintained for over 12 months during which they continue to display spontaneous seizures. A similar phenotype with spontaneous seizures has been observed in mice having 129/Sv, C3HeB/FeJ, C57BL/6 X 129/Sv, and 129/Sv X N:NIHS-BC backgrounds.
004820 C57BL/6J-Kcne12J/J
Repository-Cryopreserved
View strain phenotype and additional information on the Neuroscience Mutagenesis Facility web page for Kcne12J entry.
005748 C57BL/6J-Kcnq1vtg-3J/J
Repository-Cryopreserved
View strain phenotype and additional information on the Neuroscience Mutagenesis Facility web page for Kcnq1vtg-3J entry.
004587 C57BL/6J-Szt1/FrkJ
Repository-Cryopreserved
View strain phenotype and additional information on the Neuroscience Mutagenesis Facility web page for Szt1 entry.

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